Have “Numerous Controlled Transmission Studies” Proven Viral Contagion? The claim that they have is often repeated, but when scrutinized, many of the most-cited studies fall short of meeting rigorous scientific standards for proving viral transmission.

Some Historical Context: The 1918 Influenza Studies

·         The U.S. Navy and Public Health Service conducted multiple experiments trying to transmit influenza:

o    Healthy volunteers were exposed to mucus, breath, and even injected blood of flu victims.

o    Not one case of illness was successfully induced, even with close contact.

·         These studies failed to prove human-to-human transmission, despite their best efforts. This is largely ignored today.

Modern COVID Transmission Models

·         Much of what is labeled “transmission proof” involves:

o    PCR tests (which detect genetic material—not infectious particles)

o    Animal studies with forced inoculation (intranasal drops, injection into lungs—not natural exposure)

o    Computer simulations and contact-tracing assumptions.

Problem:
No modern study has demonstrated clean, reproducible infection from a purified, isolated virus introduced naturally (e.g., via breath or contact) in humans.

Even studies in animals (e.g., ferrets, hamsters) involve:

·         Use of lab-enhanced viral suspensions

·         Additives (e.g., trypsin)

·         Cell cultures that do not mimic real-world conditions.

These are proxy methods, not proof of natural airborne contagion.

 What About Koch’s Postulates?

Koch's postulates were the gold standard for proving causality between a pathogen and disease. Here's a refresher:

Koch’s Postulate

Challenge with Viruses

1. Pathogen must be found in all cases

SARS-CoV-2 detected via PCR, not isolated from every sick person

2. Must be isolated and grown in pure culture

Viruses aren’t grown in pure culture—they require host cells

3. Must cause disease when introduced to healthy host

Ethical limits prevent this in humans; animal proxies are flawed

4. Re-isolated and shown to be same

Re-isolation often based on PCR—not full virus particles

Modern virology claims to have “updated” these postulates using molecular biology, but this introduces new issues:

·         PCR detects snippets of RNA, not whole viruses.

·         Genetic sequences are often assembled in silico (digitally) rather than from whole purified virions.

·         No visual confirmation of isolated, infectious SARS-CoV-2 presented in studies meeting all isolation criteria without contamination.

What Would Real Proof Look Like?

To truly prove a virus causes disease via natural transmission, you would need:

1.      Purified virus (not cultured with antibiotics, trypsin, monkey cells, or genetic guesswork)

2.      Introduced to a healthy host via natural means (e.g., breathing, skin contact)

3.      Induction of identical symptoms

4.      Re-isolation of the same virus from the sick host

5.      Consistent reproduction of results across multiple independent trials

This standard has not been met for SARS-CoV-2, influenza, or HIV. Instead, what is called “proof” often relies on synthetic gene sequences, contaminated cultures, and circular logic (“we assume it’s contagious because people got sick”).

THE REAL QUESTION: What Are We Actually Observing?

·         People get sick, yes—but is it transmission of a germ, or shared exposure to toxins, fear, EMFs, poor air, or bad food?

·         Illness often spreads in families or cities, but so do environmental factors.

·         Could what we label "flu season" be seasonal detox cycles triggered by temperature, stress, and nutrient deficiencies?

Terrain theorists argue: When you clean the terrain, illness disappears—even when surrounded by the “sick.”

CONCLUSION: 
SCIENCE REQUIRES DEMONSTRABLE PROOF, NOT ASSUMED DOGMA

So when public health officials say:

“Controlled studies prove viruses like SARS-CoV-2 and influenza are contagious…”

They are typically referring to:

·         Synthetic experiments

·         Non-natural exposure methods

·         PCR-based correlations, not direct causation

When rigorously examined, these do not meet the burden of proof for demonstrating viral contagion in real-world, human-to-human contact.

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